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18-Oct-2016 9:00 AM EDT
A New Look at Vitamin D Challenges the Current View of Its Benefits
Buck Institute for Research on Aging

Research in C. elegans shows the popular supplement engages longevity genes to increase lifespan and prevent the accumulation of toxic proteins linked to many age-related diseases

Released: 16-Jun-2016 8:00 AM EDT
Pre and Post Testing Show Reversal of Memory Loss From Alzheimer’s Disease in Ten Patients
Buck Institute for Research on Aging

Small trial from the Buck Institute and UCLA succeeds using systems approach to memory disorders.

Released: 20-Nov-2013 2:00 PM EST
Aging Impacts Epigenome in Human Skeletal Muscle
Buck Institute for Research on Aging

Our epigenome is a set of chemical switches that turn parts of our genome off and on and are impacted by environmental factors including diet, exercise and stress. Research at the Buck Institute reveals that aging also effects the epigenome in human skeletal muscle. The study provides a method to study sarcopenia, the degenerative loss of muscle mass that begins in middle age.

18-Nov-2013 3:40 PM EST
USC and Buck Institute Launch Nation's First PhD Program in Biology of Aging
Buck Institute for Research on Aging

To tackle the rising prevalence of age-related diseases and the challenges and opportunities presented by a growing elderly population, the USC Davis School of Gerontology and the Buck Institute for Research on Aging today announced a joint Ph.D. program in the Biology of Aging, the first in the nation.

16-Oct-2013 4:00 PM EDT
Major Alzheimer’s Risk Factor Linked to Red Wine Target
Buck Institute for Research on Aging

The major genetic risk factor for Alzheimer’s disease, present in about two-thirds of people who develop it, is ApoE4, the cholesterol-carrying protein that about a quarter of us are born with. But one of the unsolved mysteries of AD is how ApoE4 causes this risk. Researchers at the Buck Institute have found a link between ApoE4 and SirT1, an “anti-aging protein” that is targeted by resveratrol, present in red wine.

Released: 14-Oct-2013 4:00 PM EDT
Halloween Candy Spooks Aging Digestive Systems! Research in Fruit Flies Helps Explain Why
Buck Institute for Research on Aging

Have you ever wondered why young children can eat bags of Halloween candy and feel fine the next day – compared to adults who experience all sorts of agony following the same junk food binge? Evolution and a gene called Foxo may be to blame.

21-Jun-2013 8:00 AM EDT
Research in Fruit Flies Provides New Insight Into Barrett's Esophagus
Buck Institute for Research on Aging

Research focused on the regulation of the adult stem cells that line the gastrointestinal tract of Drosophila suggests new models for the study of Barrett’s esophagus. Barrett’s esophagus is a condition in which the cells of the lower esophagus transform into stomach-like cells. In most cases this transformation has been thought to occur directly from chronic acid indigestion. A new study suggests a change in stem cell function for this transformation.

Released: 10-Jun-2013 1:00 PM EDT
Lifespan-Extending Drug Given Late in Life Reverses Age-Related Heart Disease in Mice
Buck Institute for Research on Aging

Mice suffering from age-related heart disease saw a significant improvement in cardiac function after treatment with the FDA-approved drug rapamycin for just three months. Research at the Buck Institute shows how rapamycin impacts mammalian tissues, providing functional insights and possible benefits for a drug that can extend lifespan in mice as much as 14 percent. Researchers at the Mayo clinic are now recruiting seniors with cardiac artery disease for a clinical trial involving the drug.

26-Nov-2012 1:30 PM EST
Genome-Scale Study ID’s Hundreds of Drug Targets for Huntington’s
Buck Institute for Research on Aging

Scientists seeking to develop treatments for Huntington’s disease just got a roadmap that could dramatically speed their discovery process. Researchers at the Buck Institute have used RNAi technology to identify hundreds of “druggable” molecular targets linked to the toxicity associated with HD. The gene RRAS, involved in cell motility and neuronal development , was among the diverse range of modifiers identified. RRAS was revealed as a potent modulator of HD toxicity in multiple HD models.


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