Corticospinal activity is temporally coded with precise prehension movements in mice. Disrupting this patterned activity impairs movements, highlighting the critical role of corticospinal network modulation in the execution of precision movements.
A new study by Burke Neurological Institute (BNI), Weill Cornell Medicine, finds that activation of MAP2K signaling by genetic engineering or non-invasive repetitive transcranial magnetic stimulation (rTMS) promotes corticospinal tract (CST) axon sprouting and functional regeneration after spinal cord injury (SCI) in mice.
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