UCLA scientists have traced the Pavlovian response to a small cluster of brain cells -- the same neurons that go awry during Huntington’s disease, Parkinson’s disease and Tourette syndrome. The research could one day help scientists find new approaches to diagnosing and treating these neurological disorders.
An international team of scientists, led by researchers at University of California San Diego School of Medicine and University of California San Francisco, has developed a novel genetic score that allows individuals to calculate their age-specific risk of developing Alzheimer’s disease (AD), based upon genetic information.
A study led by Assistant Professor Feng Lei from National University of Singapore’s Yong Loo Lin School of Medicine has found that regular consumption of tea lowers the risk of cognitive decline in the elderly, and this is especially so for APOE e4 gene carriers who are genetically at risk of developing Alzheimer’s disease.
From human behavior such as mother/infant bonding, addiction and communication disorders to devastating brain diseases like Parkinson’s and Alzheimer’s disease, neuroscientists and other researchers from FAU’s Brain Institute are at the forefront of innovative research that will generate knowledge to benefit society.
Middle-aged people who experience temporary blood pressure drops that often cause dizziness upon standing up may be at an increased risk of developing cognitive decline and dementia 20 years later, new Johns Hopkins Bloomberg School of Public Health research suggests.
A specific protein inside cells senses threatening changes in its environment, such as heat or starvation, and triggers an adaptive response to help the cell continue to function and grow under stressful conditions, according to a new study by scientists from the University of Chicago.
Researchers have identified a new mechanism by which inflammation can spread throughout the brain after injury. This mechanism may explain the widespread and long-lasting inflammation that occurs after traumatic brain injury, and may play a role in other neurodegenerative diseases.
A study by Indiana University researchers has identified 24 compounds -- including caffeine -- with the potential to boost an enzyme in the brain shown to protect against dementia. The research appeared March 7 in the journal Scientific Reports.
Specialized nerve cells, known as somatostatin-expressing (Sst) interneurons, in the outer part of the mammalian brain (or cerebral cortex) — play a key role in controlling how information flows in the brain when it is awake and alert. This is the finding of a study published online in Science March 2 by a team of neuroscientists at NYU Langone Medical Center and its Neuroscience Institute.
A Mayo Clinic research team has found a new gene mutation that may be a risk factor for late-onset Alzheimer’s disease in African-Americans.
NeuroVision Imaging LLC (“NeuroVision”) today announced its participation in a new substudy with investigators at the University of California San Diego School of Medicine (UC San Diego) and the University of Southern California (USC) to be part of the landmark Anti-Amyloid Treatment in Asymptomatic Alzheimer’s (or “A4”) clinical trial. The purpose of the A4 study is to test whether a new investigational treatment that may reduce beta-amyloid accumulation in the brain can also slow memory loss caused by Alzheimer’s disease.
Donepezil, a medication that is approved to treat people with Alzheimer’s disease, should not be prescribed for people with mild cognitive impairment without a genetic test.
Researchers at the University of Illinois at Chicago College of Nursing have developed a free mobile app for individuals suffering from dementia, their families and caregivers, as a way to improve the quality-of-life, well-being and knowledge of the disease that affects nearly 48 million people worldwide.
Rush University Medical Center has launched a unique, cohort study called Latino Core to learn about the aging process and risk factors for Alzheimer’s disease in older Latino adults.
Cedars-Sinai neuroscientists have uncovered processes involved in how the human brain creates and maintains short-term memories. This study is the first clear demonstration of precisely how human brain cells work to create and recall short-term memories. Confirmation of this process and the specific brain regions involved is a critical step in developing meaningful treatments for memory disorders that affect millions of Americans.
Researchers from the UGR and UMA have designed a technique that aims to model high-level data abstractions to make computers learn to differentiate the brain of a healthy person from that of an ill person by extracting the affected regions.
Two Kennesaw State University scientists have received a total of $737,364 in National Science Foundation and National Institutes of Health grants for developmental biology research into autism and birth defects.
The latest study on hormone therapy and Alzheimer’s disease shows no relationship between taking the drugs and whether you may develop the disease years later. Some previous studies have shown that hormone therapy may increase the risk of the disease, while others have shown that it may reduce the risk. The new study was published in the February 15, 2017, online issue of Neurology®, the medical journal of the American Academy of Neurology.
Calcium in the mitochondria -- the energy factory of cells -- may be one of the keys to understanding and treating Alzheimer's disease and dementia. Researchers at Temple University have now identified how an imbalance of calcium ions in the mitochondria may contribute to cell death and, specifically, neurodegeneration in brain cells during Alzheimer's and dementia. The findings could eventually point to new therapies for preventing or delaying these diseases. The team will present its work during the 61st Meeting of the Biophysical Society.
Currently, the predominant theory behind Alzheimer’s disease is the “amyloid hypothesis,” which states that abnormally increased levels of amyloid beta (Aβ) peptides outside of brain cells produce a variety of low molecular weight Aβ aggregates that are toxic to the nervous system. These Aβ aggregates interact directly with target cells and lead to cell death. During the Biophysical Society’s meeting, being held Feb. 11-15, 2017, Antonio De Maio will present his work hunting for the specific mechanisms behind Aβ-induced toxicity to cells, or cytoxicity.
RSS
Medicine keyboard_arrow_right
Sciencekeyboard_arrow_right
Life keyboard_arrow_right
Business keyboard_arrow_right
Journal News keyboard_arrow_right
By Location keyboard_arrow_right
Meetings, Grants, and Events keyboard_arrow_right