Tumor-Suppressing Protein May Be Novel Target in PAH Therapy

Embargoed Until May 19, 2:45 p.m. CT

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Dacia Morris
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ATS Office 212-315-8620 (until May 16)

Session: A97 Station to Station: Unraveling the Molecular Pathogenesis of PAH
Abstract Presentation Time:  Sunday, May 19, 2:45 p.m. CT
Location: Room C147, 148 and154 (Level 1), Kay Bailey Hutchison Convention Center Dallas

TUMOR-SUPPRESSING PROTEIN MAY BE NOVEL TARGET IN PAH THERAPY

ATS 2019, Dallas, TX ─ In addition to suppressing tumors, the protein tuberous sclerosis complex 2 (TSC2) may play a role in preventing or treating pulmonary arterial hypertension (PAH), according to research presented at ATS 2019. Researchers found that a deficiency of this protein produced dysfunctional molecules unable to support cell structure and function, resulted in thickening of pulmonary blood vessels and led to PAH.

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CONTACT FOR MEDIA

Yuanjun Shen, PhD 
[email protected]