Newswise — City of Industry, CA – January 20, 2013 – Whether one reads news alerts from the National Institutes of Health (NIH), science reporting from The Washington Post or stays in touch with wellness developments by perusing The Natural Food Merchandiser, there is a resounding consensus among key influencers in the health and wellness community that cognitive health, dementia and Alzheimer’s disease (AD) will continue to be major challenges for consumers in 2014.1 Five million Americans suffer with Alzheimer’s disease.2 More than 93,000 U.S. Alzheimer’s patients die each year. The good news is that Alzheimer’s is at the forefront of biomedical research, and progress is being made in identifying possible nutrients that may actually restore brain health.
In surveys conducted by the American Association of Retired People and ASA-MetLife Foundation, nearly 9 out of 10 consumers believe it is possible to improve cognitive fitness, and more than 4 out of 5 people older than 50 say “staying mentally sharp” is their No. 1 concern. A deteriorating mental state concerns baby boomers more than death itself.3
According to the National Alzheimer’s Association:
• Alzheimer's is the most common form of dementia, a general term for memory loss and other intellectual abilities serious enough to interfere with daily life. Alzheimer's disease accounts for 50 to 80 percent of dementia cases.
• Alzheimer's is the sixth leading cause of death in the United States.
• Today, there is a worldwide effort under way to find better ways to treat the disease, delay its onset, and prevent it from developing.4
The Role of Magnesium-L-Threonate, Magtein®, In Cognitive Health
Magnesium is an essential nutrient long associated with regulating blood pressure, metabolism, and calcium in the body.5
However, recent peer-reviewed scientific studies have demonstrated that Magnesium-L-Threonate or Magtein, not other forms of magnesium, exerts substantial positive effects on brain synapes in a mouse model of AD, actually restoring aging brains to their youthful conditions.6 The early scientific research on Magtein was conducted by a team at the Massachusetts Institute of Technology, including the 1987 Nobel Laureate in Biology.
NIH notes that while different types of drugs are being used to treat Alzheimer’s disease, Parkinson’s dementia, and certain other forms of dementia, these medications may improve symptoms, but none can slow down, halt or reverse progressive damage to the brain.7 Magtein is the only nutrient that has been shown in animal models to reverse the conditions of old Alzheimer’s animals (add reference VI). It has also been shown in Humans to significantly enhance cognitive functions and decrease symptoms of cognitive impairments.
Dr. Guosong Liu, one of the world’s leading cognitive health researchers said, “The body of our peer-reviewed and published work underscores that magnesium threonate can help maintain healthy brain activity. There is no doubt that magnesium threonate has dramatic effects in preventing synapse loss and reversing memory decline in mice with Alzheimer’s disease. More exciting, though, are the implications of this study for the potential for treating AD in humans.”
Editor’s Note: A double blind, placebo controlled human trial is completed and the exciting data is expected to be published in a peer-reviewed journal later this year. For interviews with Dr. Liu, please contact Pamela Stewart, Essenza Communications, 303-570-1678.
A complete discussion of the peer-reviewed and published science behind Magtein is available at www.magtein.com/thescience.html.
1. http://newsinhealth.nih.gov/issue/Jan2014/Feature1; http://www.washingtonpost.com/national/health-science/10-nutrients-that-can-lift-your-spirits/2014/01/14/05f4e514-7a4d-11e3-b1c5-739e63e9c9a7_story.html; and http://newhope360.com/supplements/rethinking-brain-health?cid=nl_npi_daily&&sfvc4enews=42&cl=article_1
6. Elevation of Brain Magnesium Prevents and Reverses Cognitive Deficits and Synaptic Loss in Alzheimer's Disease Mouse Model”, Li W., Yu J., Liu Y., Huang X., Abumaria N., Zhu Y., Huang X., Xiong W., Ren C., Liu X., Chui D., and Liu G., Journal of Neuroscience, 2013. 33(19): p8423-41.
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